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Dr. Clancy McKenzie relaxes in India
If one percent of those research dollars were spent identifying peak age of origin and age range of origin for each psychiatric symptom or disorder, there no longer would be a question as to origin of mental and emotional problems
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The difference
The most important difference between the combat veteran and the person with schizophrenia is that while the veteran flashes back to the brain structures he was using as an adult, the person with Schizophrenia flashes back to the brain structures he was using as an infant.
These are the earlier developmental regions of the brain, the parts of the brain we were using before we became so smart. The parts of the brain we were using before we learned to talk, or even the parts of the brain we were using before we learned to walk – depending on how early the original trauma
occurred.
These earlier brain structures are the ones that produce more of the neurotransmitters involved in the disease process, such as Dopamine. And when reactivated, they produce more! Is this not obvious? Why has no one suspected this? Why has everyone looked only from the opposite direction and wondered how Dopamine causes schizophrenia?
Likewise there is a shift of brain activity away from the later developmental structures. Remember the full-grown man sitting in the middle of the floor screaming Mommy! Mommy!” Was he using the part of his brain that developed in adult life? Obviously not! And what happens to a part of the body that becomes less active? It atrophies. Commonly expressed: If you don't use it you lose it.” So the brain atrophies as the result of the disease process.
So why hasn't anyone even entertained the thought that schizophrenia causes brain atrophy? Why do the only look at brain changes and wonder how these cause schizophrenia? Why isn't disuse atrophy the first thing researchers question? With any other part of the body that atrophies, one thinks of disuse atrophy first.
I have long puzzled over the reason why such obvious questions never were considered. Instead, researchers look at the multitude of biological changes and take this as evidence of biological cause, when common sense would dictate that it more likely represents evidence of biological results of an underlying problem. It stands to reason that if biological change is the result of the disease process, then biological change should be present in great abundance.
One reason for the myopia could be the large amount of funds devoted to the search for biological cause. The powers to be have 970 million dollars allocated for the year 2,000, largely to find the biological cause of schizophrenia. Would they be interested in doing something other than what already is bringing in 970 million dollars? More on NIMH later.
Aside from the search for biological cause, there is yet another area of investigation that continues to take center stage over and over again. This is the search for genetic cause. According to Kaplan and Saddock, a leading psychiatric resource book, nearly half of the chromosomes have been implicated in a genetic cause for schizophrenia. Several times each year, for as many decades as I remember, we have seen headlined in the newspapers: "Scientists at Zippo Ivy League University identify the gene that causes schizophrenia.” Same story each time. Only the name of the university and the identity of the chromosome differ.
Dear reader: If such a gene truly were identified, why would they come out with a different one several times each year? If one percent of those research dollars were spent identifying peak age of origin and age range of origin for each psychiatric symptom or disorder, there no longer would be a question as to origin of mental and emotional problems.
The truth of the matter is that if we combine genetic factors plus familial factors (traits run in families as well as in the genes) – only ten percent of persons with schizophrenia have a first degree relative with the disorder. It is a fact that some, or possibly even most, of these ten percent have nothing to do with heredity, since familial factors invariably are included among genetic studies as though they were genetic.
This does not mean that I disagree with genetic predisposition, but contrast the ten percent with our finding of delayed posttraumatic stress disorder. Delayed posttraumatic stress disorder can not occur without an original trauma. This is a one-to-one ratio!
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